IL-1/inhibitory κB kinase ε–induced glycolysis augment epithelial effector function and promote allergic airways disease

Author:

Qian Xi,Aboushousha Reem,van de Wetering Cheryl,Chia Shi B.,Amiel Eyal,Schneider Robert W.,van der Velden Jos L.J.,Lahue Karolyn G.,Hoagland Daisy A.,Casey Dylan T.,Daphtary Nirav,Ather Jennifer L.,Randall Matthew J.,Aliyeva Minara,Black Kendall E.,Chapman David G.,Lundblad Lennart K.A.,McMillan David H.,Dixon Anne E.,Anathy Vikas,Irvin Charles G.,Poynter Matthew E.,Wouters Emiel F.M.,Vacek Pamela M.,Henket Monique,Schleich Florence,Louis Renaud,van der Vliet Albert,Janssen-Heininger Yvonne M.W.

Funder

National Institutes of Health (NIH)

National Institute of Allergy and Infectious Diseases

National Center for Research Resources

VLC CoBRE

VCIID CoBRE

National Heart, Lung, and Blood Institute

Publisher

Elsevier BV

Subject

Immunology,Immunology and Allergy

Reference67 articles.

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3. Type 2 inflammation in asthma—present in most, absent in many;Fahy;Nat Rev Immunol,2015

4. The cell biology of asthma;Erle;J Cell Biol,2014

5. Inflammation and metabolic disorders;Hotamisligil;Nature,2006

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