Aspirin-exacerbated respiratory disease: Mediators and mechanisms of a clinical disease-Reference-Cited by-同舟云学术

Aspirin-exacerbated respiratory disease: Mediators and mechanisms of a clinical disease

Published:2017-03 Issue:3 Volume:139 Page:764-766
ISSN:0091-6749
Container-title:Journal of Allergy and Clinical Immunology
language:en
Short-container-title:Journal of Allergy and Clinical Immunology

Author:

Cahill Katherine N.,Boyce Joshua A.

Publisher

Elsevier BV

Subject

Immunology,Immunology and Allergy

Reference10 articles.

1. Aspirin-exacerbated respiratory disease—new prime suspects;Laidlaw;N Engl J Med,2016

2. Overexpression of leukotriene C4 synthase in bronchial biopsies from patients with aspirin-intolerant asthma;Cowburn;J Clin Invest,1998

3. Cysteinyl leukotriene overproduction in aspirin-exacerbated respiratory disease is driven by platelet-adherent leukocytes;Laidlaw;Blood,2012

4. Prostaglandin E2 deficiency causes a phenotype of aspirin sensitivity that depends on platelets and cysteinyl leukotrienes;Liu;Proc Natl Acad Sci U S A,2013

5. Airway responsiveness to leukotriene C4 (LTC4), leukotriene E4 (LTE4) and histamine in aspirin sensitive asthmatic subjects;Christie;Eur Respir J,1993

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