Diminished allergic disease in patients with STAT3 mutations reveals a role for STAT3 signaling in mast cell degranulation

Author:

Siegel Andrea M.,Stone Kelly D.,Cruse Glenn,Lawrence Monica G.,Olivera Ana,Jung Mi-yeon,Barber John S.,Freeman Alexandra F.,Holland Steven M.,O'Brien Michelle,Jones Nina,Wisch Laura B.,Kong Heidi H.,Desai Avanti,Farber Orly,Gilfillan Alasdair M.,Rivera Juan,Milner Joshua D.

Publisher

Elsevier BV

Subject

Immunology,Immunology and Allergy

Reference32 articles.

1. STAT3 mutations in the hyper-IgE syndrome;Holland;N Engl J Med,2007

2. Dominant-negative mutations in the DNA-binding domain of STAT3 cause hyper-IgE syndrome;Minegishi;Nature,2007

3. Hyper-IgE syndrome update;Sowerwine;Ann N Y Acad Sci,2012

4. Extreme hyperimmunoglobulinemia E and undue susceptibility to infection;Buckley;Pediatrics,1972

5. Prevalence of IgE-mediated food allergy among children with atopic dermatitis;Eigenmann;Pediatrics,1998

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