Influence of pH on the prostacyclin (PGI2) mediated inhibition of platelet function
Author:
Publisher
Elsevier BV
Subject
General Medicine
Reference18 articles.
1. Arterial walls generate from prostaglandin endoperoxides a substance, (prostaglandin x) which relaxes strips of mesenteric and coeliac arterial and inhibits platelet aggregation;Bunting;Prostaglandins,1976
2. The chemical structure of prostaglandin x, (prostacyclin);Johnson;Prostaglandins,1976
3. Human arterial and venous tissues generate prostacyclin (prostaglandin x) a potent inhibitor of platelet aggregation;Moncada;Lancet,1977
4. Effect of prostacyclin (prostaglandin x) on cyclic AMP concentration in human platelets;Tateson;Prostaglandins,1977
5. Relative potency of prostacyclin, prostaglandin E, and D2 as inhibitors of platelet aggregation in several species;Moncada;J. Physiol,1977
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2. Peripartum Substitution of Inhaled for Intravenous Prostacyclin in a Patient with Primary Pulmonary Hypertension;Anesthesiology;2004-06-01
3. Inhaled prostacyclin is safe, effective, and affordable in patients with pulmonary hypertension, right heart dysfunction, and refractory hypoxemia after cardiothoracic surgery;The Journal of Thoracic and Cardiovascular Surgery;2004-04
4. Effect of chitosan on lingual hemostasis in rabbits with platelet dysfunction induced by epoprostenol;Journal of Oral and Maxillofacial Surgery;1992-01
5. Plasma protein binding and biologic half-life of prostacyclin in steady state sickle cell disease;Thrombosis Research;1991-12
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