Hypothesis: Prostaglandins mediate defective glucose recognition in diabetes mellitus
Author:
Publisher
Elsevier BV
Subject
General Medicine
Reference33 articles.
1. The glucose receptor: a defective mechanism in diabetes mellitus distinct from the beta adrenergic receptor;Robertson;Journal of Clinical Investigation,1973
2. Augmented insulin response to glucose after secretion priming in diabetic subjects;Lerner;Journal of Clinical Endocrinology and Metabolism,1979
3. Neurendocrine recognition of glucose: the glucoreceptor hypothesis and the diabetic syndrome;Porte,1977
4. Relationships between fasting plasma glucose levels and insulin secretion during intravenous glucose tolerance tests;Brunzell;Journal of Clinical Endocrinology and Metabolism,1976
5. Abnormal growth hormone response to ingestion of glucose in juvenile diabetics;Yde;Acta Medica Scandinavica,1969
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4. Starvation decreases insulin secretion, prostaglandin E2 production and phospholipase A2 activity in rat pancreatic islets;Journal of Endocrinology;1990-03
5. Arachidonic acid metabolite regulation of insulin secretion;Diabetes / Metabolism Reviews;1986
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