Patterns of FGF-23, DMP1, and MEPE expression in patients with chronic kidney disease-Reference-Cited by-同舟云学术

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Patterns of FGF-23, DMP1, and MEPE expression in patients with chronic kidney disease

Published:2009-12 Issue:6 Volume:45 Page:1161-1168
ISSN:8756-3282
Container-title:Bone
language:en
Short-container-title:Bone

Author:

Pereira Renata C.,Jűppner Harald,Azucena-Serrano Carlos E.,Yadin Ora,Salusky Isidro B.,Wesseling-Perry Katherine

Publisher

Elsevier BV

Subject

Histology,Physiology,Endocrinology, Diabetes and Metabolism

Reference34 articles.

1. Loss of DMP1 causes rickets and osteomalacia and identifies a role for osteocytes in mineral metabolism;Feng;Nat. Genet.,2006

2. Phosphorylated acidic serine-aspartate-rich MEPE-associated motif peptide from matrix extracellular phosphoglycoprotein inhibits phosphate regulating gene with homologies to endopeptidases on the X-chromosome enzyme activity;Liu;J. Endocrinol.,2007

3. Circulating concentration of FGF-23 increases as renal function declines in patients with chronic kidney disease, but does not change in response to variation in phosphate intake in healthy volunteers;Larsson;Kidney Int.,2003

4. Relationship between plasma FGF-23 concentration and bone mineralization in children with renal failure on peritoneal dialysis;Wesseling-Perry;J. Clin. Endocrinol. Metab.,2009

5. Definition, evaluation, and classification of renal osteodystrophy: a position statement from Kidney Disease: Improving Global Outcomes (KDIGO);Moe;Kidney Int.,2006

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