Effect of gene dose and parental origin on bone histomorphometry in X-linked Hyp mice
Author:
Publisher
Elsevier BV
Subject
Histology,Physiology,Endocrinology, Diabetes and Metabolism
Reference35 articles.
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3. Mammalian membrane metallopeptidases: NEP, ECE, KELL and PEX;Turner;FASEB J.,1997
4. cDNA cloning of the murine Pex gene implicated in X-linked hypophosphatemia and evidence for expression in bone;Du;Genomics,1996
5. Pex/PEX tissue distribution and evidence for a deletion in the 3′ region of the Pex gene in X-linked hypophosphatemic mice;Beck;J. Clin. Invest.,1997
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1. The contribution of a novel PHEX gene mutation to X-linked hypophosphatemic rickets: a case report and an analysis of the gene mutation dosage effect in a rat model;Frontiers in Endocrinology;2023-12-05
2. Mineralized tissues in hypophosphatemic rickets;Pediatric Nephrology;2019-08-08
3. Marked alterations in the structure, dynamics and maturation of growth plate likely explain growth retardation and bone deformities of young Hyp mice;Bone;2018-11
4. X-linked hypophosphatemia and growth;Reviews in Endocrine and Metabolic Disorders;2017-01-27
5. Phosphate homeostasis and genetic mutations of familial hypophosphatemic rickets;Journal of Pediatric Endocrinology and Metabolism;2015-01-01
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