Mef2c regulates bone mass through Sost-dependent and -independent mechanisms-Reference-Cited by-同舟云学术

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Mef2c regulates bone mass through Sost-dependent and -independent mechanisms

Published:2024-02 Issue: Volume:179 Page:116976
ISSN:8756-3282
Container-title:Bone
language:en
Short-container-title:Bone

Author:

Morfin Cesar,Sebastian Aimy,Wilson Stephen P.,Amiri Beheshta,Murugesh Deepa K.,Hum Nicholas R.,Christiansen Blaine A.,Loots Gabriela G.

Funder

National Institutes of Health

U.S. Department of Energy

Lawrence Livermore National Laboratory

Publisher

Elsevier BV

Subject

Histology,Physiology,Endocrinology, Diabetes and Metabolism

Reference80 articles.

1. Primary osteoporosis in postmenopausal women;Ji;Chronic Dis. Transl. Med.,2015

2. An estimate of the worldwide prevalence and disability associated with osteoporotic fractures;Johnell;Osteoporos. Int.,2006

3. Fracture prevention with zoledronate in older women with osteopenia;Reid;N. Engl. J. Med.,2018

4. Parathyroid hormone stimulates the bone apposition rate independently of its resorptive action: differential effects of intermittent and continuous administration;Tam;Endocrinology,1982

5. Effect of parathyroid hormone (1-34) on fractures and bone mineral density in postmenopausal women with osteoporosis;Neer;N. Engl. J. Med.,2001

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1. Direct activation of PI3K in osteoblasts and osteocytes strengthens murine bone through sex-specific actions on cortical surfaces;Journal of Bone and Mineral Research;2024-07-03

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