Serum fibroblast growth factor 23, serum iron and bone mineral density in premenopausal women
Author:
Funder
National Institutes of Health
Scottish Rite of Indianapolis Foundation
Publisher
Elsevier BV
Subject
Histology,Physiology,Endocrinology, Diabetes and Metabolism
Reference38 articles.
1. FGF-23 is a potent regulator of vitamin D metabolism and phosphate homeostasis;Shimada;J. Bone Miner. Res.,2004
2. Fibroblast growth factor-23 mutants causing familial tumoral calcinosis are differentially processed;Larsson;Endocrinology,2005
3. Iron modifies plasma FGF23 differently in autosomal dominant hypophosphatemic rickets and healthy humans;Imel;J. Clin. Endocrinol. Metab.,2011
4. Iron deficiency drives an autosomal dominant hypophosphatemic rickets (ADHR) phenotype in fibroblast growth factor-23 (Fgf23) knock-in mice;Farrow;Proc. Natl. Acad. Sci. U. S. A.,2011
5. Increased circulatory level of biologically active full-length FGF-23 in patients with hypophosphatemic rickets/osteomalacia;Yamazaki;J. Clin. Endocrinol. Metab.,2002
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