Constitutively active Stat5b signaling confers tolerogenic functions to dendritic cells of NOD mice and halts diabetes progression-Reference-Cited by-同舟云学术

Constitutively active Stat5b signaling confers tolerogenic functions to dendritic cells of NOD mice and halts diabetes progression

Published:2017-01 Issue: Volume:76 Page:63-74
ISSN:0896-8411
Container-title:Journal of Autoimmunity
language:en
Short-container-title:Journal of Autoimmunity

Author:

Zerif Echarki,Maalem Aida,Gaudreau Simon,Guindi Chantal,Ramzan Muhammad,Véroneau Steeve,Gris Denis,Stankova Jana,Rola-Pleszczynski Marek,Mourad Walid,Dupuis Gilles,Amrani Abdelaziz

Funder

Canadian Institute of Health Research (CIHR)

Publisher

Elsevier BV

Subject

Immunology,Immunology and Allergy

Reference61 articles.

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4. Mouse models for the study of autoimmune type 1 diabetes: a NOD to similarities and differences to human disease;Driver;Semin. Immunopathol.,2011

5. Genetic protection from the inflammatory disease type 1 diabetes in humans and animal models;Todd;Immunity,2001

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3. Dendritic Cells and Their Immunotherapeutic Potential for Treating Type 1 Diabetes;International Journal of Molecular Sciences;2022-04-28

4. Advanced Glycation End Products: New Clinical and Molecular Perspectives;International Journal of Environmental Research and Public Health;2021-07-06

5. Elucidating the Role of Ezh2 in Tolerogenic Function of NOD Bone Marrow-Derived Dendritic Cells Expressing Constitutively Active Stat5b;International Journal of Molecular Sciences;2020-09-04