Spontaneous recovery from early glomerular inflammation is associated with resistance to anti-GBM glomerulonephritis: tolerance and autoimmune tissue injury
Author:
Publisher
Elsevier BV
Subject
Immunology,Immunology and Allergy
Reference40 articles.
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2. Glomerulonephritis induced in sheep by injection of heterologous GBM and Freund's complete adjuvant;Steblay;J. Exp. Med.,1962
3. Isolation of the specific glomerular basement membrane antigen involved in Goodpasture syndrome;Wieslander;Proc. Natl. Acad. Sci. USA,1984
4. Specificity of Goodpasture autoantibodies for the recombinant noncollagenous domains of human type IV collagen;Neilson;J. Biol. Chem.,1993
5. The α3 chain of type IV collagen induces autoimmune Goodpasture syndrome;Kalluri;Proc. Natl. Acad. Sci. USA,1994
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1. ID3 may protect mice from anti-GBM glomerulonephritis by regulating the differentiation of Th17 and Treg cells;Molecular Medicine Reports;2017-10-04
2. CD8αα+MHC Class II+ Cell with the Capacity To Terminate Autoimmune Inflammation Is a Novel Antigen-Presenting NK-like Cell in Rats;The Journal of Immunology;2016-10-31
3. Differentiating Glomerular Inflammation from Fibrosis in a Bone Marrow Chimera for Rat Anti-Glomerular Basement Membrane Glomerulonephritis;American Journal of Nephrology;2015
4. Peripheral blood CD8αα + CD11c + MHC-II + CD3 - cells attenuate autoimmune glomerulonephritis in rats;Kidney International;2014-05
5. Goodpasture's disease: A report of ten cases and a review of the literature;Autoimmunity Reviews;2013-09
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