Regulation of the anorectic drug recognition site during glucoprivic feeding
Author:
Publisher
Elsevier BV
Subject
General Neuroscience
Reference50 articles.
1. Glucostatic regulation of (+)[3H]amphetamine binding in the hypothalamus: correlation with Na+K+ATPase activity;Angel,1985
2. Regulation of [3H]mazindol binding to subhypothalamic areas: Involvement in glucoprivic feeding;Angel;Brain Res. Bull.,1986
3. Characterization of [3H]mazindol binding in the rat brain: sodium sensitive binding correlates with the anorectic potencies of phenylethylamines;Angel;J. Neurochem.,1987
4. Hypothalamic [3H]mazindol binding correlates with the anorectic properties of phenylethylamines;Angel;Eur. J. Pharmacol.,1985
5. Site of action of anorectic drugs: Glucoprivic- versus food deprivation-induced feeding;Angel;Pharmacol. Biochem. Behav.,1987
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1. Current and potential drugs for treatment of obesity;ENDOCR REV;1999
2. Current and Potential Drugs for Treatment of Obesity;Endocrine Reviews;1999-12-01
3. Protection by 2-Deoxy-d-glucose against β,β′-Iminodipropionitrile-Induced Neurobehavioral Toxicity in Mice;Experimental Neurology;1999-07
4. Is There Appetite after GLP-1 and PACAP?;Annals of the New York Academy of Sciences;1998-12
5. Increased potency and binding of mazindol to putative brain anorectic receptors in obesity-prone rats;Brain Research;1994-12
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