CD8+iTregs attenuate glomerular endothelial cell injury in lupus-prone mice through blocking the activation of p38 MAPK and NF-κB-Reference-Cited by-同舟云学术

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CD8+iTregs attenuate glomerular endothelial cell injury in lupus-prone mice through blocking the activation of p38 MAPK and NF-κB

Published:2018-11 Issue: Volume:103 Page:133-143
ISSN:0161-5890
Container-title:Molecular Immunology
language:en
Short-container-title:Molecular Immunology

Author:

Liu Ya,Deng Weijuan,Meng Qiaoyun,Qiu Xiaonan,Sun Dong,Dai Chun

Funder

Jiangsu Provincial Natural Science Foundation

National Natural Science Foundation of China

China Postdoctoral Science Fund

12th “Liu Da Ren Cai Gao Feng”of Jiangsu Province, China

Jiangsu Provincial Medical Talent

Publisher

Elsevier BV

Subject

Molecular Biology,Immunology

Reference66 articles.

1. Systemic lupus erythematosus biomarkers: the challenging quest;Arriens;Rheumatology (Oxford),2017

2. Detection of nuclear factor-kappaB in IgA nephropathy using Southwestern histochemistry;Ashizawa;Am. J. Kidney Dis. Off. J. Natl. Kidney Found.,2003

3. Pathology and pathogenesis of vascular injury in systemic lupus erythematosus. Interactions of inflammatory cells and activated endothelium;Belmont;Arthritis Rheum.,1996

4. p53 induces NF-kappaB activation by an IkappaB kinase-independent mechanism involving phosphorylation of p65 by ribosomal S6 kinase 1;Bohuslav;J. Biol. Chem.,2004

5. Leukocyte adhesion molecules and kidney diseases;Brady;Kidney Int.,1994

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