CLEC5A (MDL-1) is a novel PU.1 transcriptional target during myeloid differentiation-Reference-Cited by-同舟云学术

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CLEC5A (MDL-1) is a novel PU.1 transcriptional target during myeloid differentiation

Published:2011-01 Issue:4 Volume:48 Page:714-719
ISSN:0161-5890
Container-title:Molecular Immunology
language:en
Short-container-title:Molecular Immunology

Author:

Batliner Jasmin,Mancarelli Maria Michela,Jenal Mathias,Reddy Venkateshwar A.,Fey Martin F.,Torbett Bruce E.,Tschan Mario P.

Funder

Swiss National Science Foundation

3100A0-118276

Bern University Research Foundation

Werner and Hedy Berger-Janser Foundation of Cancer Research

NIH

HL091219

DK54938

Joyce Klein Stock Gift

Marlies-Schwegler Foundation

Ursula-Hecht-Foundation for Leukemia Research

Bernese Foundation of Cancer Research

Publisher

Elsevier BV

Subject

Molecular Biology,Immunology

Reference21 articles.

1. A rapid micropreparation technique for extraction of DNA-binding proteins from limiting numbers of mammalian-cells;Andrews;Nucleic Acids Res.,1991

2. Differential regulation of DAP12 and molecules associated with DAP12 during host responses to mycobacterial infection;Aoki;Infect. Immun.,2004

3. Expression and functional role of MDL-1 (CLEC5A) in mouse myeloid lineage cells;Aoki;J. Leukoc. Biol.,2009

4. Myeloid DAP12-associating lectin (MDL)-1 is a cell surface receptor involved in the activation of myeloid cells;Bakker;Proc. Natl. Acad. Sci. U.S.A.,1999

5. DAP12: a key accessory protein for relaying signals by natural killer cell receptors;Campbell;Int. J. Biochem. Cell Biol.,1999

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