Hepatocytes paradoxically affect intrahepatic IFN-γ production in autoimmune hepatitis due to Gal-9 expression and TLR2/4 ligand release-Reference-Cited by-同舟云学术

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Hepatocytes paradoxically affect intrahepatic IFN-γ production in autoimmune hepatitis due to Gal-9 expression and TLR2/4 ligand release

Published:2020-07 Issue: Volume:123 Page:106-115
ISSN:0161-5890
Container-title:Molecular Immunology
language:en
Short-container-title:Molecular Immunology

Author:

Mo Ran,Feng Xin-Xia,Wu Ya-Nan,Wang Han,He Yong-Pei,Sun Huan-Huan,Guo Fang,Chen Qian,Yan Wei,Li Pei-Yuan,Liu Mei,Zhang Gui-Mei,Tian De-An,Feng Zuo-Hua

Funder

National Natural Science Foundation of China

Publisher

Elsevier BV

Subject

Molecular Biology,Immunology

Reference61 articles.

1. Hepatocyte entry leads to degradation of autoreactive CD8 T cells;Benseler;Proc. Natl. Acad. Sci. U.S.A.,2011

2. Role of primary intrahepatic T-cell activation in the’ liver tolerance effect’;Bertolino;Immunol. Cell Biol.,2002

3. Multifaceted death receptor 3 signaling-promoting survival and triggering death;Bittner;FEBS Lett.,2017

4. The site of primary T cell activation is a determinant of the balance between intrahepatic tolerance and immunity;Bowen;J. Clin. Invest.,2004

5. In vivo expression of interleukin-37 reduces local and systemic inflammation in concanavalin A-induced hepatitis;Bulau;ScientificWorldJournal.,2011

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5. Construction and function analysis of the LncRNA-miRNA-mRNA competing endogenous RNA network in autoimmune hepatitis;BMC Medical Genomics;2022-12-25

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