Role of early lectin pathway activation in the complement-mediated killing of Trypanosoma cruzi

Author:

Cestari Igor dos S.,Krarup Anders,Sim Robert B.,Inal Jameel M.,Ramirez Marcel I.

Funder

Royal Society Travel Grant

Publisher

Elsevier BV

Subject

Molecular Biology,Immunology

Reference42 articles.

1. GPI-anchored glycoconjugates from Trypanosoma cruzi trypomastigotes are recognized by lytic anti-alpha-galactosyl antibodies isolated from patients with chronic Chagas’ disease;Almeida;Braz. J. Med. Biol. Res.,1994

2. Complement-mediated lysis of Trypanosoma cruzi trypomastigotes by human anti-alpha-galactosyl antibodies;Almeida;J. Immunol.,1991

3. Leishmania (Viannia) braziliensis: interaction of mannose-binding lectin with surface glycoconjugates and complement activation. An antibody-independent defence mechanism;Ambrosio;Parasite Immunol.,2005

4. Trypanosoma cruzi surface mucins: host-dependent coat diversity;Buscaglia;Nat. Rev. Microbiol.,2006

5. Complement C2 receptor inhibitor trispanning confers an increased ability to resist complement-mediated lysis in Trypanosoma cruzi;Cestari;J. Infect. Dis.,2008

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