IL36G genetic variant is independently associated with susceptibility, severity and joint involvement in psoriasis

Author:

Moreira Cássio Rafael,de Alcântara Camila Cataldi,Flauzino Tamires,Martin Ligia Marcia Mario,Lozovoy Marcell Alysson Batisti,Reiche Edna Maria Vissoci,Simão Andréa Name Colado

Funder

Conselho Nacional de Desenvolvimento Científico e Tecnológico

Coordenação de Aperfeiçoamento de Pessoal de Nível Superior

Ministry of Education

Publisher

Elsevier BV

Subject

Molecular Biology,Immunology

Reference34 articles.

1. Psoriasis;Boehncke;Lancet,2015

2. Brazil. IBGE. Brazilian Institute of Geography and Statistics Characteristics of the population and households: results of the universe. https://ww2.ibge.gov.br/english/estatistica/populacao/censo2010/caracteristicas_da_populacao/default_ caracteristicas_da_populacao.shtm, 2011.

3. Sequence variants in the genes for the interleukin-23 receptor (IL23R) and its ligand (IL12B) confer protection against psoriasis;Capon;Hum. Genet,2017

4. Overview of the molecular determinants contributing to the expression of Psoriasis and Psoriatic Arthritis phenotypes;Caputo;J. Cell Mol. Med,2020

5. A large-scale genetic association study confirms IL12B and leads to the identification of IL23R as psoriasis-risk genes;Cargill;Am. J. Hum. Genet,2007

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