ATP binding by NLRP7 is required for inflammasome activation in response to bacterial lipopeptides-Reference-Cited by-同舟云学术

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ATP binding by NLRP7 is required for inflammasome activation in response to bacterial lipopeptides

Published:2015-10 Issue:2 Volume:67 Page:294-302
ISSN:0161-5890
Container-title:Molecular Immunology
language:en
Short-container-title:Molecular Immunology

Author:

Radian Alexander D.,Khare Sonal,Chu Lan H.,Dorfleutner Andrea,Stehlik Christian

Funder

National Institutes of Health

Skin Disease Research Center

American Heart Association

Arthritis Foundation

Vietnam Education Foundation

Publisher

Elsevier BV

Subject

Molecular Biology,Immunology

Reference56 articles.

1. Structural localization of disease-associated sequence variations in the NACHT and LRR domains of PYPAF1 and NOD2;Albrecht;FEBS Lett.,2003

2. The NLRP3 inflammasome is released as a particulate danger signal that amplifies the inflammatory response;Baroja-Mazo;Nat. Immunol.,2014

3. Differential splicing of the apoptosis-associated speck like protein containing a caspase recruitment domain (ASC) regulates inflammasomes;Bryan;J. Inflamm. (Lond.),2010

4. Prion-like polymerization underlies signal transduction in antiviral immune defense and inflammasome activation;Cai;Cell,2014

5. An updated view on the structure and function of PYRIN domains;Chu;Apoptosis,2015

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