Inflammatory pathways in spondyloarthritis
Author:
Publisher
Elsevier BV
Subject
Molecular Biology,Immunology
Reference122 articles.
1. IL-23 is critical for induction of arthritis, osteoclast formation, and maintenance of bone mass;Adamopoulos;Journal of Immunology (Baltimore, MD),2011
2. A murine transmembrane tumor necrosis factor (TNF) transgene induces arthritis by cooperative p55/p75 TNF receptor signaling;Alexopoulou;European Journal of Immunology,1997
3. Pathogenesis of spondyloarthritis: autoimmune or autoinflammatory?;Ambarus;Current Opinion in Rheumatology,2012
4. In situ analysis of interleukin-23- and interleukin-12-positive cells in the spine of patients with ankylosing spondylitis;Appel;Arthritis & Rheumatism,2013
5. Analysis of IL-17(+) cells in facet joints of patients with spondyloarthritis suggests that the innate immune pathway might be of greater relevance than the Th17-mediated adaptive immune response;Appel;Arthritis Research & Therapy,2011
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