Warm-blood cardioplegic arrest induces selective mitochondrial translocation of protein kinase Cε followed by interaction with 6.1 inwardly rectifying potassium channel subunit in viable myocytes overexpressing urocortin-Reference-Cited by-同舟云学术

Warm-blood cardioplegic arrest induces selective mitochondrial translocation of protein kinase Cε followed by interaction with 6.1 inwardly rectifying potassium channel subunit in viable myocytes overexpressing urocortin

Published:2009-11 Issue:5 Volume:138 Page:1213-1221
ISSN:0022-5223
Container-title:The Journal of Thoracic and Cardiovascular Surgery
language:en
Short-container-title:The Journal of Thoracic and Cardiovascular Surgery

Author:

Chen-Scarabelli Carol,Faggian Giuseppe,Yuan Zhaokan,Tessari Maddalena,Rungatscher Alessio,Di Rezze Justin,Scarabelli Gabriele M.,Abounit Kadija,McCauley Roy,Saravolatz Louis,Mazzucco Alessandro,Scarabelli Tiziano M.

Publisher

Elsevier BV

Subject

Cardiology and Cardiovascular Medicine,Pulmonary and Respiratory Medicine,Surgery

Reference26 articles.

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4. Ultrastructural changes in myocardium during mild hypothermic retrograde blood cardioplegia;Rainio;Scand Cardiovasc J,1998

5. Endothelial apoptosis is induced by serum of patients after cardiopulmonary bypass;Aebert;Eur J Cardiothorac Surg,2000

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2. Urocortin Induces Phosphorylation of Distinct Residues of Signal Transducer and Activator of Transcription 3 (STAT3) via Different Signaling Pathways;Medical Science Monitor Basic Research;2019-05-10

3. Do urocortins have a role in treating cardiovascular disease?;Drug Discovery Today;2019-01

4. Diabetic hearts have lower basal urocortin levels that fail to increase after cardioplegic arrest: Association with increased apoptosis and postsurgical cardiac dysfunction;The Journal of Thoracic and Cardiovascular Surgery;2014-11

5. Targeting Urocortin Signaling Pathways to Enhance Cardioprotection: Is It Time to Move from Bench to Bedside?;Cardiovascular Drugs and Therapy;2013-07-04