Overexpression of catalase or Bcl-2 delays or prevents alterations in phospholipid metabolism during glucocorticoid-induced apoptosis in WEHI7.2 cells-Reference-Cited by-同舟云学术

Overexpression of catalase or Bcl-2 delays or prevents alterations in phospholipid metabolism during glucocorticoid-induced apoptosis in WEHI7.2 cells

Published:2003-10 Issue:3 Volume:1642 Page:149-162
ISSN:0167-4889
Container-title:Biochimica et Biophysica Acta (BBA) - Molecular Cell Research
language:en
Short-container-title:Biochimica et Biophysica Acta (BBA) - Molecular Cell Research

Author:

Tome Margaret E.,Lutz Norbert W.,Briehl Margaret M.

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology

Reference79 articles.

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4. Transcriptional transactivation functions localized to the glucocorticoid receptor N terminus are necessary for steroid induction of lymphocyte apoptosis;Dieken;Mol. Cell. Biol.,1992

5. Identification of the activation-labile gene: a single point mutation in the human glucocorticoid receptor presents as two distinct receptor phenotypes;Ashraf;Mol. Endocrinol.,1993

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