Acidic environment activates inflammatory programs in fibroblasts via a cAMP–MAPK pathway-Reference-Cited by-同舟云学术

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Acidic environment activates inflammatory programs in fibroblasts via a cAMP–MAPK pathway

Published:2015-02 Issue:2 Volume:1853 Page:299-307
ISSN:0167-4889
Container-title:Biochimica et Biophysica Acta (BBA) - Molecular Cell Research
language:en
Short-container-title:Biochimica et Biophysica Acta (BBA) - Molecular Cell Research

Author:

Riemann A.,Ihling A.,Thomas J.,Schneider B.,Thews O.,Gekle M.

Funder

Deutsche Krebshilfe

BMBF (ProNet-T3 Ta-04)

Wilhelm-Roux program of the Medical School, University Halle-Wittenberg

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology

Reference68 articles.

1. Causes and consequences of tumour acidity and implications for treatment;Stubbs;Mol. Med. Today,2000

2. Acidosis activation of the proton-sensing GPR4 receptor stimulates vascular endothelial cell inflammatory responses revealed by transcriptome analysis;Dong;PLoS ONE,2013

3. Acidosis downregulates platelet haemostatic functions and promotes neutrophil proinflammatory responses mediated by platelets;Etulain;Thromb. Haemost.,2012

4. Extracellular acidosis promotes neutrophil transdifferentiation to MHC class II-expressing cells;Pliyev;Cell. Immunol.,2011

5. Low extracellular pH stimulates the production of IL-1β by human monocytes;Jancic;Cytokine,2012

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