DNA damage-induced apoptosis suppressor (DDIAS), a novel target of NFATc1, is associated with cisplatin resistance in lung cancer

Author:

Im Joo-Young,Lee Kang-Woo,Won Kyoung-Jae,Kim Bo-Kyung,Ban Hyun Seung,Yoon Sung-Hoon,Lee Young-Ju,Kim Young-Joo,Song Kyung-Bin,Won Misun

Funder

National Research Foundation (NRF)

Korea Research Council of Fundamental Science and Technology

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology

Reference42 articles.

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3. Overexpression of c-myc in pancreatic cancer caused by ectopic activation of NFATc1 and the Ca2+/calcineurin signaling pathway;Buchholz;EMBO J.,2006

4. NFAT: ubiquitous regulator of cell differentiation and adaptation;Horsley;J. Cell Biol.,2002

5. Constitutive activation of signal transducers and activators of transcription 3 correlates with cyclin D1 overexpression and may provide a novel prognostic marker in head and neck squamous cell carcinoma;Masuda;Cancer Res.,2002

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