Activation of PERK-eIF2α-ATF4-CHOP axis triggered by excessive ER stress contributes to lead-induced nephrotoxicity-Reference-Cited by-同舟云学术

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Activation of PERK-eIF2α-ATF4-CHOP axis triggered by excessive ER stress contributes to lead-induced nephrotoxicity

Published:2019-04 Issue:4 Volume:1866 Page:713-726
ISSN:0167-4889
Container-title:Biochimica et Biophysica Acta (BBA) - Molecular Cell Research
language:en
Short-container-title:Biochimica et Biophysica Acta (BBA) - Molecular Cell Research

Author:

Wang Min-Ge,Fan Rui-Feng,Li Wen-Hui,Zhang Dong,Yang Du-Bao,Wang Zhen-Yong,Wang Lin

Funder

National Natural Science Foundation of China

Shandong Province Natural Science Foundation

Funds of Shandong “Double Tops” Program

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology

Reference42 articles.

1. Lead-related nephrotoxicity: a review of the epidemiologic evidence;Ekong;Kidney Int.,2006

2. Autophagy blockade and lysosomal membrane permeabilization contribute to lead-induced nephrotoxicity in primary rat proximal tubular cells;Song;Cell Death Dis.,2017

3. Effects of selenium-lead interaction on the gene expression of inflammatory factors and selenoproteins in chicken neutrophils;Li;Ecotoxicol. Environ. Saf.,2017

4. The antagonistic effect of selenium on lead-induced apoptosis via mitochondrial dynamics pathway in the chicken kidney;Jin;Chemosphere,2017

5. Lead accumulation and metallothionein content in female rats of different ages and generations after daily intake of Pb-contaminated food;Nascimento;Environ. Toxicol. Pharmacol.,2016

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