Pro-metastatic functions of Notch signaling is mediated by CYR61 in breast cells-Reference-Cited by-同舟云学术

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Pro-metastatic functions of Notch signaling is mediated by CYR61 in breast cells

Published:2020-04 Issue:2-3 Volume:99 Page:151070
ISSN:0171-9335
Container-title:European Journal of Cell Biology
language:en
Short-container-title:European Journal of Cell Biology

Author:

Ilhan Mustafa,Kucukkose Cansu,Efe Eda,Gunyuz Zehra Elif,Firatligil Burcu,Dogan Hulya,Ozuysal Mustafa,Yalcin-Ozuysal Ozden

Funder

Scientific and Technological Research Council of Turkey

Publisher

Elsevier BV

Subject

Cell Biology,General Medicine,Histology,Pathology and Forensic Medicine

Reference69 articles.

1. Triple negative breast cancer initiating cell subsets differ in functional and molecular characteristics and in gamma-secretase inhibitor drug responses;Azzam;EMBO Mol. Med.,2013

2. Notch-1 induces epithelial-mesenchymal transition consistent with cancer stem cell phenotype in pancreatic cancer cells;Bao;Cancer Lett.,2011

3. Notch activation stimulates migration of breast cancer cells and promotes tumor growth;Bolos;Breast Cancer Res.,2013

4. Hypoxia potentiates Notch signaling in breast cancer leading to decreased E-cadherin expression and increased cell migration and invasion;Chen;Br. J. Cancer,2010

5. Regulation of breast cancer metastasis by Runx2 and estrogen signaling: the role of SNAI2;Chimge;Breast Cancer Res.,2011

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