Crosstalk between Epidermal Growth Factor Receptors (EGFR) and integrins in resistance to EGFR tyrosine kinase inhibitors (TKIs) in solid tumors-Reference-Cited by-同舟云学术

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Crosstalk between Epidermal Growth Factor Receptors (EGFR) and integrins in resistance to EGFR tyrosine kinase inhibitors (TKIs) in solid tumors

Published:2020-05 Issue:4 Volume:99 Page:151083
ISSN:0171-9335
Container-title:European Journal of Cell Biology
language:en
Short-container-title:European Journal of Cell Biology

Author:

Javadi Samira,Zhiani Mina,Mousavi Mir Ali,Fathi Mojtaba

Funder

Zanjan University of Medical Sciences

Publisher

Elsevier BV

Subject

Cell Biology,General Medicine,Histology,Pathology and Forensic Medicine

Reference147 articles.

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2. Stimulation of β1-integrin function by epidermal growth factor and heregulin-β has distinct requirements for erbB2 but a similar dependence on phosphoinositide 3-OH kinase;Adelsman;Mol. Biol. Cell,1999

3. Increasing cancer-specific gene expression by targeting overexpressed α5β1 integrin and upregulated transcriptional activity of NF-κB;Adil;Mol. Pharm.,2014

4. Src-induced de-regulation of E-cadherin in colon cancer cells requires integrin signalling;Avizienyte;Nat. Cell Biol.,2002

5. Epithelial to Mesenchymal Transition promotes breast cancer progression via a fibronectin-dependent STAT3 signaling pathway;Balanis;J. Biol. Chem.,2013

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