Upregulation of paxillin and focal adhesion signaling follows Dystroglycan Complex deletions and promotes a hypertensive state of differentiation-Reference-Cited by-同舟云学术

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Upregulation of paxillin and focal adhesion signaling follows Dystroglycan Complex deletions and promotes a hypertensive state of differentiation

Published:2011-02 Issue:2-3 Volume:90 Page:249-260
ISSN:0171-9335
Container-title:European Journal of Cell Biology
language:en
Short-container-title:European Journal of Cell Biology

Author:

Sen Shamik,Tewari Manorama,Zajac Allison,Barton Elisabeth,Sweeney H. Lee,Discher Dennis E.

Publisher

Elsevier BV

Subject

Cell Biology,General Medicine,Histology,Pathology and Forensic Medicine

Reference78 articles.

1. Genetic compensation for sarcoglycan loss by integrin {alpha}7{beta}1 in muscle;Allikian;J. Cell Sci.,2004

2. Relaxin restores altered ileal spontaneous contractions in dystrophic (mdx) mice;Baccari;Ann. N.Y. Acad. Sci.,2005

3. A web-accessible complete transcriptome of normal human and DMD muscle;Bakay;Neuromuscul. Disord.,2002

4. Paxillin: adapting to change;Brown;Physiol. Rev.,2004

5. Transgenic expression of {alpha}7{beta}1 integrin maintains muscle integrity, increases regenerative capacity, promotes hypertrophy, and reduces cardiomyopathy in dystrophic mice;Burkin;Am. J. Pathol.,2005

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