Cholesterol-dependent molecular mechanisms contribute to cationic amphiphilic drugs’ prevention of silica-induced inflammation

Author:

Kendall Rebekah L.,Holian Andrij

Funder

National Institute of General Medical Sciences

National Institute of Environmental Health Sciences

Publisher

Elsevier BV

Subject

Cell Biology,General Medicine,Histology,Pathology and Forensic Medicine

Reference62 articles.

1. Ro of endosomal membrane lipids and NPC2 in cholesterol transfer and membrane fusion;Abdul-Hammed;J. Lipid Res.,2010

2. Methods for the quantification of lysosomal membrane permeabilization: a hallmark of lysosomal cell death;Aits;Methods Cell Biol.,2015

3. Attenuation of the lysosomal death pathway by lysosomal cholesterol accumulation;Appelqvist;Am. J. Pathol.,2011

4. Sensitivity to lysosome-dependent cell death is directly regulated by lysosomal cholesterol content;Appelqvist;PLoS One,2012

5. Role of lysosomes in silica-induced inflammasome activation and inflammation in absence of MARCO;Biswas;J. Immunol. Res,2014

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