Low-dose aspirin (ASA) renders human platelets more vulnerable to inhibition of aggregation by prostacyclin (PGI2)-Reference-Cited by-同舟云学术

Low-dose aspirin (ASA) renders human platelets more vulnerable to inhibition of aggregation by prostacyclin (PGI2)

Published:1983-06 Issue:2 Volume:11 Page:131-142
ISSN:0262-1746
Container-title:Prostaglandins, Leukotrienes and Medicine
language:en
Short-container-title:Prostaglandins, Leukotrienes and Medicine

Author:

Philp R.B.,Paul M.L.

Publisher

Elsevier BV

Subject

Endocrinology,Physiology,Biochemistry

Reference26 articles.

1. Human arterial and venous tissues generate prostacyclin (prostaglandin X), a potent inhibitor of platelet aggregation;Moncada;Lancet,1977

2. Prostacyclin and thromboxane generating systems in rabbits pretreated with aspirin;Batista;Pharmacol. Res. Commun.,1978

3. Philp RB, Paul ML, Killackey JJ, Killackey BA. The influence of dose, time of administration, body temperature and salicylate kinetics on the anti-thrombosic action of acetylsalicylic acid (ASA) in male rats. Haemostasis, in press.

4. In vitro tests of platelet function and platelet inhibiting drugs;Philp,1981

5. Biostatistical Analysis;Zar,1974

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5. Response of thromboxane B2, malondialdehyde and platelet sensitivity to 3 weeks low-dose aspirin (ASA) in healthy volunteers;Thrombosis Research;1989-02