A case of severe asthenozoospermia: a novel sperm tail defect of possible genetic origin identified by electron microscopy and immunocytochemistry-Reference-Cited by-同舟云学术

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A case of severe asthenozoospermia: a novel sperm tail defect of possible genetic origin identified by electron microscopy and immunocytochemistry

Published:2011-01 Issue:1 Volume:95 Page:289.e11-289.e16
ISSN:0015-0282
Container-title:Fertility and Sterility
language:en
Short-container-title:Fertility and Sterility

Author:

Collodel Giulia,Federico Maria Grazia,Pascarelli Nicola Antonio,Geminiani Michela,Renieri Tommaso,Moretti Elena

Publisher

Elsevier BV

Subject

Obstetrics and Gynaecology,Reproductive Medicine

Reference21 articles.

1. Genetic sperm defects and consanguinity;Baccetti;Hum Reprod,2001

2. Sperm pathology: a step beyond descriptive morphology. Origin, characterization and fertility potential of abnormal sperm phenotypes in infertile men;Chemes;Hum Reprod Update,2003

3. An ultrastructural and immunocytochemical study of a rare genetic sperm tail defect that causes infertility in humans;Baccetti;Fertil Steril,2004

4. Abnormal elongation of midpiece, absence of axoneme and outer dense fibers at principal piece level, supernumerary microtubules: a sperm defect of possible genetic origin?;Moretti;Fertil Steril,2008

5. Fertilizing ability of immotile spermatozoa after intracytoplasmic sperm injection;Nijs;Hum Reprod,1996

Cited by 10 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. ICSI outcomes for infertile men with severe or complete asthenozoospermia;Basic and Clinical Andrology;2022-04-05

2. Nonneoplastic Diseases of the Testis;Urologic Surgical Pathology;2020

3. CCDC9 is identified as a novel candidate gene of severe asthenozoospermia;Systems Biology in Reproductive Medicine;2019-09-10

4. EIF4G1 is a novel candidate gene associated with severe asthenozoospermia;Molecular Genetics & Genomic Medicine;2019-07-03

5. A familial study of twins with severe asthenozoospermia identified a homozygousSPAG17mutation by whole-exome sequencing;Clinical Genetics;2017-09-04

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