Phosphate turnover of phosphatidylinositol in resting and thrombin-stimulated platelets
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology
Reference41 articles.
1. Tight coupling of thrombin-induced acid hydrolase secretion and phosphatidate synthesis to receptor occupancy in human platelets
2. Thrombin-induced hydrolysis of phosphatidylinositol in human platelets.
3. The fatty acid composition of phosphatidylinositol from thrombin-stimulated human platelets.
4. Inositol lipids, phosphatidate and diacylglycerol share stearoylarachidonoylglycerol as a common backbone in thrombin-stimulated human platelets
5. The rapid polyphosphoinositide metabolism may be a triggering event for thrombin-mediated stimulation of human platelets
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1. Psychotropic drugs interfere with the tight coupling of polyphosphoinositide cycle metabolites in human platelets: A result of receptor-independent drug intercalation in the plasma membrane?;Biochimica et Biophysica Acta (BBA) - Biomembranes;2008-10
2. The stimulatory effects of cationic amphiphilic drugs on human platelets treated with thrombin;Biochemical Pharmacology;2000-11
3. Chlorpromazine and human platelet glycerolipid metabolism: precursor specificity and significance of drug–platelet interaction time;Biochemical Pharmacology;1999-05
4. Elevation of cyclic AMP decreases phosphoinositide turnover and inhibits thrombin-induced secretion in human platelets;Biochimica et Biophysica Acta (BBA) - Lipids and Lipid Metabolism;1998-11
5. Analysis of GTP-binding proteins, phosphoproteins, and cytosolic calcium in functional heterogeneous human blood platelet subpopulations;Biochemical Pharmacology;1997-11
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