Myocardial Galectin-3 Expression Is Associated with Remodeling of the Pressure-Overloaded Heart and May Delay the Hypertrophic Response without Affecting Survival, Dysfunction, and Cardiac Fibrosis-Reference-Cited by-同舟云学术

Myocardial Galectin-3 Expression Is Associated with Remodeling of the Pressure-Overloaded Heart and May Delay the Hypertrophic Response without Affecting Survival, Dysfunction, and Cardiac Fibrosis

Published:2016-05 Issue:5 Volume:186 Page:1114-1127
ISSN:0002-9440
Container-title:The American Journal of Pathology
language:en
Short-container-title:The American Journal of Pathology

Author:

Frunza Olga,Russo Ilaria,Saxena Amit,Shinde Arti V.,Humeres Claudio,Hanif Waqas,Rai Vikrant,Su Ya,Frangogiannis Nikolaos G.

Funder

American Heart Association

National Institutes of Health

Publisher

Elsevier BV

Subject

Pathology and Forensic Medicine

Reference53 articles.

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4. Galectin-3 is induced in rheumatoid arthritis synovial fibroblasts after adhesion to cartilage oligomeric matrix protein;Neidhart;Ann Rheum Dis,2005

5. Galectin-3 functions as an adhesion molecule to support eosinophil rolling and adhesion under conditions of flow;Rao;J Immunol,2007

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