Co-Exposure to Cigarette Smoke and Alcohol Decreases Airway Epithelial Cell Cilia Beating in a Protein Kinase Cε-Dependent Manner-Reference-Cited by-同舟云学术

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Co-Exposure to Cigarette Smoke and Alcohol Decreases Airway Epithelial Cell Cilia Beating in a Protein Kinase Cε-Dependent Manner

Published:2012-08 Issue:2 Volume:181 Page:431-440
ISSN:0002-9440
Container-title:The American Journal of Pathology
language:en
Short-container-title:The American Journal of Pathology

Author:

Wyatt Todd A.,Sisson Joseph H.,Allen-Gipson Diane S.,McCaskill Michael L.,Boten Jessica A.,DeVasure Jane M.,Bailey Kristina L.,Poole Jill A.

Funder

National Institute of Environmental Health Sciences

National Institutes of Health

U.S. Department of Veterans Affairs

National Institute on Alcohol Abuse and Alcoholism

Publisher

Elsevier BV

Subject

Pathology and Forensic Medicine

Reference38 articles.

1. Vital signs: current cigarette smoking among adults aged >or=18 years — United States, 2009;MMWR Morb Mortal Wkly Rep,2010

2. Long-term cigarette smoke exposure in a mouse model of ciliated epithelial cell function;Simet;Am J Respir Cell Mol Biol,2010

3. Mucociliary clearance is enhanced in rat models of cigarette smoke and lipopolysaccharide-induced lung disease;Coote;Exp Lung Res,2004

4. Chronic ethanol downregulates PKA activation and ciliary beating in bovine bronchial epithelial cells;Wyatt;Am J Physiol Lung Cell Mol Physiol,2001

5. Smoke exposure exacerbates an ethanol-induced defect in mucociliary clearance of Streptococcus pneumoniae;Vander Top;Alcohol Clin Exp Res,2005

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