Activation of AMP-Activated Protein Kinase Prevents TGF-β1–Induced Epithelial-Mesenchymal Transition and Myofibroblast Activation-Reference-Cited by-同舟云学术

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Activation of AMP-Activated Protein Kinase Prevents TGF-β1–Induced Epithelial-Mesenchymal Transition and Myofibroblast Activation

Published:2015-08 Issue:8 Volume:185 Page:2168-2180
ISSN:0002-9440
Container-title:The American Journal of Pathology
language:en
Short-container-title:The American Journal of Pathology

Author:

Thakur Sachin,Viswanadhapalli Suryavathi,Kopp Jeffrey B.,Shi Qian,Barnes Jeffrey L.,Block Karen,Gorin Yves,Abboud Hanna E.

Funder

National Institute of Diabetes and Digestive and Kidney Diseases

U.S. Department of Veterans Affairs

Juvenile Diabetes Research Foundation International

National Institutes of Health

Publisher

Elsevier BV

Subject

Pathology and Forensic Medicine

Reference47 articles.

1. New insights into epithelial-mesenchymal transition in kidney fibrosis;Liu;J Am Soc Nephrol,2010

2. Common and unique mechanisms regulate fibrosis in various fibroproliferative diseases;Wynn;J Clin Invest,2007

3. TGF-β1 → SMAD/p53/USF2 → PAI-1 transcriptional axis in ureteral obstruction-induced renal fibrosis;Samarakoon;Cell Tissue Res,2012

4. Adenosine monophosphate-activated protein kinase (AMPK) as a new target for antidiabetic drugs: a review on metabolic, pharmacological and chemical considerations;Gruzman;Rev Diabet Stud,2009

5. The mTOR pathway is highly activated in diabetic nephropathy and rapamycin has a strong therapeutic potential;Mori;Biochem Biophys Res Commun,2009

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