PERK Activation Preserves the Viability and Function of Remyelinating Oligodendrocytes in Immune-Mediated Demyelinating Diseases-Reference-Cited by-同舟云学术

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PERK Activation Preserves the Viability and Function of Remyelinating Oligodendrocytes in Immune-Mediated Demyelinating Diseases

Published:2014-02 Issue:2 Volume:184 Page:507-519
ISSN:0002-9440
Container-title:The American Journal of Pathology
language:en
Short-container-title:The American Journal of Pathology

Author:

Lin Yifeng,Huang Guangcun,Jamison Stephanie,Li Jin,Harding Heather P.,Ron David,Lin Wensheng

Funder

National Multiple Sclerosis Society

National Institutes of Health

Publisher

Elsevier BV

Subject

Pathology and Forensic Medicine

Reference58 articles.

1. Remyelination in the CNS: from biology to therapy;Franklin;Nat Rev Neurosci,2008

2. Enhancing remyelination in disease–can we wrap it up?;Kotter;Brain,2011

3. From endoplasmic-reticulum stress to the inflammatory response;Zhang;Nature,2008

4. Endoplasmic reticulum stress in disorders of myelinating cells;Lin;Nat Neurosci,2009

5. Endoplasmic reticulum stress and the inflammatory basis of metabolic disease;Hotamisligil;Cell,2010

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1. Orchestrating Stress Responses in Multiple Sclerosis: A Role for Astrocytic IFNγ Signaling;International Journal of Molecular Sciences;2024-07-09

2. Oligodendrocyte‐selective deletion of the eIF2α kinase Perk/Eif2ak3 limits functional recovery after spinal cord injury;Glia;2024-04-08

3. NF-κB as an Inducible Regulator of Inflammation in the Central Nervous System;Cells;2024-03-11

4. The physiological role of the unfolded protein response in the nervous system;Neural Regeneration Research;2024-01-08

5. Potential Role of Endoplasmic Reticulum Stress in Modulating Protein Homeostasis in Oligodendrocytes to Improve White Matter Injury in Preterm Infants;Molecular Neurobiology;2024-01-05

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