Ubiquitin C-Terminal Hydrolase-L1 Activity Induces Polyubiquitin Accumulation in Podocytes and Increases Proteinuria in Rat Membranous Nephropathy-Reference-Cited by-同舟云学术

Ubiquitin C-Terminal Hydrolase-L1 Activity Induces Polyubiquitin Accumulation in Podocytes and Increases Proteinuria in Rat Membranous Nephropathy

Published:2011-05 Issue:5 Volume:178 Page:2044-2057
ISSN:0002-9440
Container-title:The American Journal of Pathology
language:en
Short-container-title:The American Journal of Pathology

Author:

Meyer-Schwesinger Catherine,Meyer Tobias N.,Sievert Henning,Hoxha Elion,Sachs Marlies,Klupp Eva-Maria,Münster Silvia,Balabanov Stefan,Carrier Lucie,Helmchen Udo,Thaiss Friedrich,Stahl Rolf A.K.

Publisher

Elsevier BV

Subject

Pathology and Forensic Medicine

Reference56 articles.

1. Stability and leakiness: opposing challenges to the glomerulus;Kriz;Kidney Int,1996

2. Podocyte biology and response to injury;Mundel;J Am Soc Nephrol,2002

3. Progression of glomerular diseases: is the podocyte the culprit?;Kriz;Kidney Int,1998

4. A frequent pathway to glomerulosclerosis: deterioration of tuft architecture-podocyte damage-segmental sclerosis;Kriz;Kidney Blood Press Res,1996

5. Cellular and molecular biology of membranous nephropathy;Couser;J Nephrol,2006

Cited by 47 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Podocyte exopher-formation as a novel pathomechanism in membranous nephropathy;2024-04-07

2. The proteasome modulates endocytosis specifically in glomerular cells to promote kidney filtration;Nature Communications;2024-03-01

3. Cytoskeleton Rearrangement in Podocytopathies: An Update;International Journal of Molecular Sciences;2024-01-04

4. The Proteasome Modulates Endocytosis in a Glomerular Cell Type Specific Modality to Secure Kidney Filtration;2023-06-16

5. Non-functional ubiquitin C-terminal hydrolase L1 drives podocyte injury through impairing proteasomes in autoimmune glomerulonephritis;Nature Communications;2023-04-13