Regulation of Nur77-TLR4/MyD88 signaling pathway is required for Ginsenoside Rc ameliorates hepatic fibrosis regression by deactivating hepatic stellate cells-Reference-Cited by-同舟云学术

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Regulation of Nur77-TLR4/MyD88 signaling pathway is required for Ginsenoside Rc ameliorates hepatic fibrosis regression by deactivating hepatic stellate cells

Published:2023-10 Issue:7 Volume:125 Page:152079
ISSN:0065-1281
Container-title:Acta Histochemica
language:en
Short-container-title:Acta Histochemica

Author:

Qin Bo-Feng,Gao Shan,Feng Qi-yuan,Chen Wei,Sun Hai-Ming,Song Jian^ORCID

Publisher

Elsevier BV

Subject

Cell Biology,Histology,General Medicine

Reference24 articles.

1. Insulin resistance and altered systemic glucose metabolism in mice lacking Nur77;Chao;Diabetes,2009

2. Deletion of TLR4 attenuates lipopolysaccharide-induced acute liver injury by inhibiting inflammation and apoptosis;Chen;Acta Pharm. Sin.,2021

3. TLR4 and CD14 trafficking and its influence on LPS-induced pro-inflammatory signaling;Ciesielska;Cell. Mol. Life Sci.,2021

4. Insight Into TLR4-mediated immunomodulation in normal pregnancy and related disorders;Firmal;Front. Immunol.,2020

5. Accelerated partial hepatectomy-induced liver cell proliferation is associated with liver injury in Nur77 knockout mice;Hu;Am. J. Pathol.,2014

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1. The roles of orphan nuclear receptor 4 group A1 and A2 in fibrosis;International Immunopharmacology;2024-09

2. Identification of (E)-1-((1H-indol-3-yl)methylene)-4-substitute-thiosemicarbazones as potential anti-hepatic fibrosis agents;Bioorganic Chemistry;2024-02

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