Subchronic exposure to acrylamide leads to pancreatic islet remodeling determined by alpha cell expansion and beta cell mass reduction in adult rats

Author:

Stošić Milena,Matavulj Milica,Marković Jelena

Funder

Ministry of Education, Science and Technological Development of the Republic of Serbia

Publisher

Elsevier BV

Subject

Cell Biology,Histology,General Medicine

Reference53 articles.

1. A histological and immunohistochemical study of beta cells in streptozotocin diabetic rats treated with caffeine;Abunasef;Folia. Histochem. Cytobiol.,2014

2. Subchronic toxicity of acrylamide administered to rats in the drinking water followed by up to 144 days of recovery;Burek;J. Environ. Pathol. Toxicol.,1980

3. Genotoxicity and carcinogenicity of acrylamide: a critical review;Carere;Ann. Ist. Super. Sanita,2006

4. Islet amyloid, increased A-cells, reduced B-cells and exocrine fibrosis: quantitative changes in the pancreas in type 2 diabetes;Clark;Diabetes Res.,1988

5. Mechanisms of pancreatic beta-cell death in type 1 and type 2 diabetes: many differences, few similarities;Cnop;Diabetes,2005

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