Administration of Levetiracetam after prolonged status epilepticus does not protect from mitochondrial dysfunction in a rodent model
Author:
Publisher
Elsevier BV
Subject
Clinical Neurology,Neurology
Reference10 articles.
1. Mitochondrial dysfunction and oxidative stress in epilepsy;Cock,2005
2. Mitochondrial dysfunction associated with neuronal death following status epilepticus in rat;Cock;Epilepsy Res.,2002
3. Levetiracetam: antiepileptic properties and protective effects on mitochondrial dysfunction in experimental status epilepticus;Gibbs;Epilepsia,2006
4. Neuronal cell death in a rat model for mesial temporal lobe epilepsy is induced by the initial status epilepticus and not by later repeated spontaneous seizures;Gorter;Epilepsia,2003
5. The role of mitochondria in epileptogenesis;Kunz;Curr. Opin. Neurol.,2002
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1. Effect of levetiracetam on DNA oxidation and glutathione content in a temporal lobe epilepsy model;Acta Neurobiologiae Experimentalis;2024-03-28
2. Efficacy of a combined anti-seizure treatment against cholinergic established status epilepticus following a sarin nerve agent insult in rats;Toxicology and Applied Pharmacology;2024-03
3. Levetiracetam combined with ACEA, highly selective cannabinoid CB1 receptor agonist changes neurogenesis in mouse brain;Neuroscience Letters;2019-03
4. Levetiracetam administration is correlated with lower mortality in patients with mitochondrial encephalomyopathy, lactic acidosis, and stroke-like episodes;Chinese Medical Journal;2019-02-05
5. Levetiracetam as an antiepileptic, neuroprotective, and hyperalgesic drug;Neurology India;2016
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