New opportunities in the management and treatment of refractory hypercholesterolemia using in vivo CRISPR-mediated genome/base editing-Reference-Cited by-同舟云学术

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New opportunities in the management and treatment of refractory hypercholesterolemia using in vivo CRISPR-mediated genome/base editing

Published:2023-12 Issue:12 Volume:33 Page:2317-2325
ISSN:0939-4753
Container-title:Nutrition, Metabolism and Cardiovascular Diseases
language:en
Short-container-title:Nutrition, Metabolism and Cardiovascular Diseases

Author:

Srivastava Rai Ajit K.^ORCID

Publisher

Elsevier BV

Subject

Cardiology and Cardiovascular Medicine,Nutrition and Dietetics,Endocrinology, Diabetes and Metabolism,Medicine (miscellaneous)

Reference128 articles.

1. The LDL receptor defect in familial hypercholesterolemia. Implications for pathogenesis and therapy;Goldstein;Med Clin North Am,1982

2. Type of LDLR mutation and the pharmacogenetics of familial hypercholesterolemia treatment;Santos;Pharmacogenomics,2015

3. Genetic architecture of familial hypercholesterolaemia;Sharifi;Curr Cardiol Rep,2017

4. Homozygous familial hypercholesterolemia;Nohara;J Atherosclerosis Thromb,2021

5. Low-density lipoproteins cause atherosclerotic cardiovascular disease. 1. Evidence from genetic, epidemiologic, and clinical studies. A consensus statement from the European Atherosclerosis Society Consensus Panel;Ference;Eur Heart J,2017

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