Pro-inflammatory effect of TWEAK/Fn14 interaction on human umbilical vein endothelial cells
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry,Biophysics
Reference28 articles.
1. Signaling by the TNF Receptor Superfamily and T Cell Homeostasis
2. Apoptosis by Death Factor
3. The CD95 (APO-1/Fas) and the TRAIL (APO-2L) Apoptosis Systems
4. TWEAK, a New Secreted Ligand in the Tumor Necrosis Factor Family That Weakly Induces Apoptosis
5. Involvement of Tweak in Interferon γ–Stimulated Monocyte Cytotoxicity
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3. Extracellular Vesicle-Associated TWEAK Contributes to Vascular Inflammation and Remodeling During Acute Cellular Rejection;JACC: Basic to Translational Science;2023-05
4. 3β-Hydroxy-5β-hydroxy-B-norcholestane-6β-carboxaldehyde (SEC-B) Induces Proinflammatory Activation of Human Endothelial Cells Associated with Nitric Oxide Production and Endothelial Nitric Oxide Synthase/Caveolin-1 Dysregulation;Antioxidants;2022-06-10
5. Comparative transcriptome analysis of human and murine choroidal neovascularization identifies fibroblast growth factor inducible-14 as phylogenetically conserved mediator of neovascular age-related macular degeneration;Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease;2022-04
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