The increased activity of a transcription factor inhibits autophagy in diabetic embryopathy
Author:
Funder
National Institutes of Health
Publisher
Elsevier BV
Subject
Obstetrics and Gynaecology
Reference42 articles.
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2. Effect of maternal diabetes on the embryo, fetus, and children: congenital anomalies, genetic and epigenetic changes and developmental outcomes;Ornoy;Birth Defects Res C Embryo Today,2015
3. Birth defects in pregestational diabetes: defect range, glycemic threshold and pathogenesis;Gabbay-Benziv;World J Diabetes,2015
4. Endoplasmic reticulum stress-induced CHOP inhibits PGC-1alpha and causes mitochondrial dysfunction in diabetic embryopathy;Chen;Toxicol Sci,2017
5. Protein kinase C-alpha suppresses autophagy and induces neural tube defects via miR-129-2 in diabetic pregnancy;Wang;Nat Commun,2017
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