Advances in revealing the molecular targets downstream of oxidative stress–induced proapoptotic kinase signaling in diabetic embryopathy
Author:
Funder
National Institutes of Health
Publisher
Elsevier BV
Subject
Obstetrics and Gynaecology
Reference124 articles.
1. SOD1 overexpression in vivo blocks hyperglycemia-induced specific PKC isoforms: substrate activation and consequent lipid peroxidation in diabetic embryopathy;Li;Am J Obstet Gynecol,2011
2. Oxidative stress-induced JNK1/2 activation triggers proapoptotic signaling and apoptosis that leads to diabetic embryopathy;Li;Diabetes,2012
3. c-Jun NH2-terminal kinase 1/2 and endoplasmic reticulum stress as interdependent and reciprocal causation in diabetic embryopathy;Li;Diabetes,2013
4. Superoxide dismutase 1 overexpression in mice abolishes maternal diabetes-induced endoplasmic reticulum stress in diabetic embryopathy;Wang;Am J Obstet Gynecol,2013
5. Trehalose prevents neural tube defects by correcting maternal diabetes-suppressed autophagy and neurogenesis;Xu;Am J Physiol Endocrinol Metab,2013
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