Negatively-charged amino acids at the peptide-binding pocket of HLA-DPB1 alleles are associated with susceptibility to anti-topoisomerase I-positive systemic sclerosis
Author:
Publisher
Elsevier BV
Subject
General Medicine,Immunology,Immunology and Allergy
Reference21 articles.
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2. The role of genetics and epigenetics in the pathogenesis of systemic sclerosis;Broen;Nat. Rev. Rheumatol.,2014
3. The HLA-DR and DQ genes control the autoimmune response to DNA topoisomerase I in systemic sclerosis (scleroderma);Kuwana;J. Clin. Invest.,1993
4. HLA-DPB1 and DPB2 are genetic loci for systemic sclerosis: a genome-wide association study in Koreans with replication in North Americans;Zhou;Arthritis Rheum.,2009
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1. Insights into origins and specificities of autoantibodies in systemic sclerosis;Current Opinion in Rheumatology;2021-09-09
2. Immunopeptidome Analysis of HLA-DPB1 Allelic Variants Reveals New Functional Hierarchies;The Journal of Immunology;2020-04-29
3. Amino acid signatures of HLA Class-I and II molecules are strongly associated with SLE susceptibility and autoantibody production in Eastern Asians;PLOS Genetics;2019-04-25
4. ANCA-Associated Vasculitis;Genetics of Rare Autoimmune Diseases;2019
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