Artificial taste avoidance memory induced by coactivation of NMDA and β-adrenergic receptors in the amygdala

Author:

Osorio-Gómez Daniel,Bermúdez-Rattoni Federico,Guzmán-Ramos Kioko

Funder

CONACyT

DGAPA PAPIIT-UNAM

FOSSIS-CONACyT

UAM-PTC-585)

Publisher

Elsevier BV

Subject

Behavioral Neuroscience

Reference63 articles.

1. Classical conditioning and sensitization share aspects of the same molecular cascade in Aplysia;Kandel;Cold Spring Harb. Symp. Quant. Biol.,1983

2. Memory formation depends on both synapse-specific modifications of synaptic strength and cell-specific increases in excitability;Lisman;Nat. Neurosci.,2018

3. Post-acquisition release of glutamate and norepinephrine in the amygdala is involved in taste-aversion memory consolidation;Guzmán-Ramos;Learn. Mem.,2012

4. Off-line concomitant release of dopamine and glutamate involvement in taste memory consolidation;Guzmán-Ramos;J. Neurochem.,2010

5. Glutamatergic activity in the amygdala signals visceral input during taste memory formation;Miranda;Proc. Natl. Acad. Sci. U. S. A.,2002

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