The T-type calcium channel antagonist, Z944, alters social behavior in Genetic Absence Epilepsy Rats from Strasbourg

Author:

Marks Wendie N.,Zabder Nadine K.,Cain Stuart M.,Snutch Terrance P.,Howland John G.ORCID

Funder

Brain Canada Multi-Investigator Research Initiative Grant

Genome British Columbia

Michael Smith Foundation for Health Research, and the Koerner Foundation

Canadian Institutes of Health Research

Canada Research Chair in Biotechnology and Genomics-Neurobiology

BC Epilepsy Society reseach award and the Cure - Taking Flight Award

Saskatchewan Health Research Foundation

University of Saskatchewan

Publisher

Elsevier BV

Subject

Behavioral Neuroscience

Reference57 articles.

1. Pathophysiological mechanisms of genetic absence epilepsy in the rat;Danober;Prog. Neurobiol,1998

2. Genetic Absence Epilepsy in Rats from Strasbourg (GAERS);Marescaux;Ital. J. Neurol. Sci.,1995

3. Genetic absence epilepsy in rats from Strasbourg--a review;Marescaux;J. Neural Transm. Suppl.,1992

4. Discharges in ventromedial frontal cortex during absence spells;Tucker;Epilepsy Behav.,2007

5. CaV3.2 drives sustained burst-firing, which is critical for absence seizure propagation in reticular thalamic neurons;Cain;Epilepsia.,2018

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