Studies of the mechanisms involved in the fate of prostacyclin (PGI2) and 06-keto-PGF1α in the pulmonary circulation
Author:
Publisher
Elsevier BV
Subject
Endocrinology,Biochemistry
Reference34 articles.
1. An enzyme isolated from arteries transforms prostaglandin endoperoxides to an unstable substance that inhibits platelet aggregation;Moncada;Nature,1976
2. A lipid peroxide inhibits the enzyme in blood vessel microsomes that generates from prostaglandin endoperoxides the substance (prostaglandin X) which prevents platelet aggregation;Moncada;Prostaglandins,1976
3. Arterial walls generate from prostaglandin endoperoxides a substance (prostaglandin X) which relaxes strips of mesenteric and coeliac arteries and inhibits platelet aggregation;Bunting;Prostaglandins,1976
4. Mechanistic studies on the biosynthesis of 6-ketoprostaglandin F1α;Pace-Asciak;Biochim. Biophys. Acta,1977
5. The chemical structure of prostaglandin X (prostacyclin);Johnson;Prostaglandins,1976
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