Protein aggregation and neurodegeneration in prototypical neurodegenerative diseases: Examples of amyloidopathies, tauopathies and synucleinopathies-Reference-Cited by-同舟云学术

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Protein aggregation and neurodegeneration in prototypical neurodegenerative diseases: Examples of amyloidopathies, tauopathies and synucleinopathies

Published:2017-08 Issue: Volume:155 Page:171-193
ISSN:0301-0082
Container-title:Progress in Neurobiology
language:en
Short-container-title:Progress in Neurobiology

Author:

Bourdenx Mathieu,Koulakiotis Nikolaos Stavros,Sanoudou Despina,Bezard Erwan,Dehay Benjamin,Tsarbopoulos Anthony

Funder

European Commission

Fondation pour la Recherche Médicale

Agence Nationale de la Recherche

LABEX BRAIN

Greek General Secretariat of Research and Technology

Publisher

Elsevier BV

Subject

General Neuroscience

Reference396 articles.

1. Mice lacking alpha-synuclein display functional deficits in the nigrostriatal dopamine system;Abeliovich;Neuron,2000

2. C-terminal inhibition of tau assembly in vitro and in Alzheimer's disease;Abraha;J. Cell Sci.,2000

3. Prions: protein aggregation and infectious diseases;Aguzzi;Physiol. Rev.,2009

4. A novel in vivo model of tau propagation with rapid and progressive neurofibrillary tangle pathology: the pattern of spread is determined by connectivity, not proximity;Ahmed;Acta Neuropathol.,2014

5. The role of oxidative stress in the pathophysiology of cerebrovascular lesions in Alzheimer's disease;Aliev;Brain Pathol.,2002

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