Dysregulation of alternative splicing underlies synaptic defects in familial amyotrophic lateral sclerosis-Reference-Cited by-同舟云学术

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Dysregulation of alternative splicing underlies synaptic defects in familial amyotrophic lateral sclerosis

Published:2023-12 Issue: Volume:231 Page:102529
ISSN:0301-0082
Container-title:Progress in Neurobiology
language:en
Short-container-title:Progress in Neurobiology

Author:

Verdile Veronica,Palombo Ramona,Ferrante Gabriele,Ferri Alberto,Amadio Susanna,Volonté Cinzia,Paronetto Maria Paola^ORCID

Funder

Associazione Italiana per la Ricerca sul Cancro

Agenzia di Ricerca per la Sclerosi Laterale Amiotrofica

Ministero della Salute

AriSLA

Fondazione Umberto Veronesi

Publisher

Elsevier BV

Subject

General Neuroscience

Reference78 articles.

1. Alternative splicing as a regulator of development and tissue identity;Baralle;Nat. Rev. Mol. Cell Biol.,2017

2. The evolutionary landscape of alternative splicing in vertebrate species;Barbosa-Morais;Science,2012

3. Ablation of the Sam68 gene impairs female fertility and gonadotropin-dependent follicle development;Bianchi;Hum. Mol. Genet,2010

4. C9orf72 loss-of-function: a trivial, stand-alone or additive mechanism in C9 ALS/FTD?;Braems;Acta Neuropathol.,2020

5. Amyotrophic lateral sclerosis. Insights from genetics;Brown;Arch. Neurol.,1997

Cited by 1 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Transcriptional profiling of human brain cortex identifies novel lncRNA-mediated networks dysregulated in amyotrophic lateral sclerosis;2024-03-19

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