Failure of dietary α-difluoromethylornithine to inhibit gastric carcinogenesis in rats after 8 weeks of treatment with N-methyl-N′-nitro-N-nitrosoguanidine and sodium chloride

Author:

Tanakamaru Zen-yo,Nishikawa Akiyoshi,Furukawa Fumio,Imazawa Takayoshi,Lee In-Seon,Kasahara Ken-ichiro,Tanaka Takuji,Takahashi Michihito

Publisher

Elsevier BV

Subject

Cancer Research,Oncology

Reference27 articles.

1. B.E. Butterworth, J.A. Popp, R.B. Conolly, T.L. Goldsworthy, Chemically induced cell proliferation in carcinogenesis, in: H. Vainio, P. Magee, D. McGregor, A.J. McMichael (Eds.), Mechanisms of Carcinogenesis in Risk Identification, IARC Scientific Publication No. 116, IARC, Lyon, 1992, pp. 279–305.

2. Systemic inhibition of tumor promoter-induced ornithine decarboxylase in 1α-hydroxyvitamin D3-treated animals;Hashiba;Cancer Res.,1987

3. Inhibition of N-butyl-N-(4-hydroxybutyl)nitrosamine-induced rat urinary bladder carcinogenesis by α-difluoromethylornithine;Homma;Cancer Res.,1987

4. Chemopreventive effects of 24R,25-dihydroxyvitamin D3, a vitamin D3 derivative, on glandular stomach carcinogenesis induced in rats by N-methyl-N′-nitro-N-nitrosoguanidine and sodium chloride;Ikezaki;Cancer Res.,1996

5. Transcriptional activation of mammalian ornithine decarboxylase during stimulated growth;Katz;Mol. Cell Biol.,1987

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